New research into cellular functions has uncovered a potential new means of controlling the effects of multiple sclerosis (MS).
Research led by the University of Adelaide has found that modification of a molecule known as PI3Kgamma, which is involved in the activation and movement of white blood cells, can be used to minimise inflammation associated with the disease.
The molecule has been shown to be crucial for the development of experimental autoimmune encephalitis, meaning that knocking it out can protect against the nervous system damage typical of multiple sclerosis.
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